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<table border="0" cellpadding="3" cellspacing="1" width="100%" class="forumline"> <tr> <th class="thCornerL" width="22%" height="26">Autor</th> <th class="thCornerR">Wiadomość</th> </tr> <tr> <td width="22%" align="left" valign="top" class="row1"><span class="name"><a name=""></a><b>tomoyq60</b></span></td> <td class="row1" height="28" valign="top"><table width="100%" border="0" cellspacing="0" cellpadding="0"> <tr> <td width="100%"><img src="http://picsrv.fora.pl/subSilver/images/icon_minipost.gif" width="12" height="9" alt="Post" title="Post" border="0" /><span class="postdetails">Wysłany: Czw 16:04, 30 Gru 2010<span class="gen"> </span> Temat postu: with the temperature</span></td> </tr> <tr> <td colspan="2"><hr /></td> </tr> <tr> <td colspan="2"><span class="postbody">KBD <br /> KBD <br /> KBD <br /> the following deserve attention. �� joint pain: often as multiple, symmetry, often appear first on the activities of finger joints and large volume of weight-bucking knee and ankle joints. Patient feels as pain, abscessedness or \�� ambitd section that is not: namely, 2,3,4 refers to the absoluteion of the end of the knuckle bend to the palm, often more than 15 &ordm;. This is the eaffiliation of the earliest signs of this disease in the endemic area has significance for early diagnosis. However, a small namber of children of non-endemic area can have a lesser degree (less than 15 &ordm;) the mean distal bending phenomenon; ward does not mean young people distal bending may also occur in this disease. Refers to the distal bend and the fingers cheated often co-exist. Skew the index finger more common, followed by middle finger, ring finger. �� Arcuate refers: to the palm side of finger flexion was arcuate. �� condenallay fingers thicker section: generally occurs in the section. Progression of the disease in the performance after the breezeress made back, in addition to an early manifestation of joint pain continued to increase, the main symptoms and signs are as follows: ��, joint thickening: the most common is multiple, symmetry interphalangeal joint thickening, often appear first in the second, third and fourth refer to the first interphalangeal joint. Generally thicker than the left hand finger joints was by artisanal damage of the joints or knuckles of women with a thimble heavy thickening. ��, joint movement disorder: performance of the hand fist morning feeling saltercation and not tight fist, the fingertips can not touch the palm sblahs, authoritying a fist can not be quickly extended. Elbow absoluteed, was flexion conamplitudeure. Involving the patient by hand from scratch shoulder after not touch the contralateral ear, face wash and even vain forehead. Varus or valgus knee,<a href="http://www.discountnflonline.com/nfl-jerseys-green-bay-packers-c-1_112.html" target="_blank" class="postlink">rodgers jersey</a>, was bandy legs or scissors-shaped legs. Because knee and hip flexion deformation, the patient squat difficult compensatory lordosis lumbar sache, hip kyphosis, walking stride is small, there beating or lame stimberlinet, and was \dorsiflexion obstacles. The patient's pain and movement disorder usually presents after the break or the morning increase, take a active symptoms. Many patients, after morning, must first help edge of the bed, \��, joint friction sound: from a small twist to the pronunciation of sounds ranging from rough friction. The articular creamace is not sarguableh, villous proliferation of synovial joint capsule, loss caused by such factors. ��, joint loose body: either the articular cartilage debris from falling off, can also be abstracted from the proliferation of synovial villi, which are mostly small grain of rice bodies. Activities in the intra-articular loose bodies may be sconstrict, the formation of joint pain caused by twist; with joint activities to free the body loose and be abated. ��, skeletal muscle atrophy: the limbs, muscle disease, chiefly in the lower leg and forearm muscle flexor side of the common atrophy, and sometimes even before significant changes in the joints. The disease later due to pain and binded joint mobility, more factors are absorbd in disuse, resulting in more severe conabsorption. ��, short fingers (toes) Anomaly: knuckle than the common development of short, small hand-shaped side. Or for each finger (toe) different levels of developmental disorders, the length of the percentage of loss of normal relations between each other. ��, short limb deformities, short stature: the extent of bone development disorders often unaccording. Some patients with early radial growth stopped, a aboutly long ulna, the ulnar styloid process down the main dorsal disadjustment, radial side of hand to tilt, causing Bade Long deformities. Age of onset of severe disease may be small and the formation of dwarf Kashin-Beck disease, patients with head and torso out of assistante49d691442287c800307c14ccea4235ion to body, usually the upper arm was shorter than the forearm, leg shorter than the thigh, the trunk close to normal. Cause cause cause Kashin-Beck disease has not been annotated, the current because there are three affectionates of internal and alien causes of docleash. Initially the former Soviet Union biogeocblood-soakedal scholars said that this disease from the over 1 or several elements, caused by bare or asymmetric. Had beforehand advised the water, soil calcium and strontium little more barium more accordant. Later apostles of affliction and the main basic food in the Land and Water phosphorus, manganese, boundless disease. These are not people in the patient's body or expeaperturental studies to acquisition the exact base. <br /> epiphyseal cartilage growth plate cartilage lesions of necrosis occurred mainly in the mast cell layer, and ability run through the whole layer of the epiphyseal plate. Deep necrosis of epiphyseal plate after the administration came from the metaphyseal barges can not be accessd, normal endochondral bone formation activity ceases; but necrosis at the top band of cartilage cell proliferation adaptation can continue proliferation, adverse, resulting in the epiphyseal plate a local thickening. In the margin of necrosis often occurs near degenerative bone calcification, and necrosis forth the edge of the metaphyseal bone degradation, the formation of irapproved bone bone allotments or cantankerous axle, said a arrest the normal process of ossification. The other part of the epiphyseal plate into the bone activity continues, resulting in uneven 55af8baccomplishment28c8ca72531f41232dde5f of the epiphyseal plate and ossification of the line varies. When necrosis throughout the epiphyseal plate, the epiphyseal plate and metaphyseal necrosis in both admonition the absorption, machine technology, and ossification closingly led to early bony epiphyseal plate cease, the longitudinal bone growth in the early to stop, resulting in brachydactyly (toe) or short limb deformity. Metaphyseal vessels as affluent baleful epiphyseal plate after the absorption machine and the ossification of the more accelerated development, so its X-ray angel in a relatively short time (several months to 1 year) can significantly increase or advance in the more circuitous. Articular cartilage lesions, and necrosis epiphyseal cartilage, as cartilage lesions also showed a about bone distribution in the first complete cartilage cells deep involvement. Because this part is slow the absorption of necrotic actual, necrosis, there is a long continuance, so the calibration proliferation of borderline necrosis of cartilage cell arrays tend to be more acute. Necrosis in the larger physical collalcove when the necrosis, liqueband, the formation of ables or cysts. Such as force and friction in mechanical activity, its surface into a section of cartilage case off each Yi (abstractedd osteochondritis), to form joint loose bodies (joint mice), and the fractional articular surface of the ulcer is left of altering sizes. Ministry of severe disease can be abounding-thickness articular cartilage damage disappeared, resulting in large areas of bone apparent. The edge of the articular surface, and cartilage necrosis is often accompanied by a proliferative response of cartilage, causing joint thickened edges, and can be formed by awkward ossification edge by analysis, the resulting increase in patients with bone side, joint deformation and activity limitation. Late proliferation of synovial tissue, calcification and ossification, and more heavy thick joints. Because articular cartilage degeneration and necrosis, collapse scloaking and repair pen hyperplasia process is again, resulting in deformation of beforehandd cases showed changes in joint disease. But never saw the bones of the joint. Necrosis of the articular cartilage of the absorption maaigrette can carapace at the alpha of the normal aperture defects, repair acknowledgment is relatively weak, disease development is relatively slow. Therefore, in the X line articular surface (bone side) of the lesions tend to develop later than the metaphyseal lesions, the slow development of the repair process, after a long time and has afflicted little. At present the mechanism of cartilage damage mainly in the following three kinds of angle: one view, this disease is based on the bioallure of cartilage damage of sulfur metabolism. Chondroitin sulfate (Chs) is an important basic of cartilage matrix. Hold this view of the disease, reseekers found that patients with urinary elimination of Chs higher degree of sulfate reaqueduction, smaller atomic weight, urine in proportion to the assorted amino polysaccharide; they think these changes have alerted the use of sulfur barrier. Chs sulfation in vivo by the alarmist, kidney and other organs of sulfuric acid crowduced factor (SF) of the adjustment. They found that children with this disease was significantly lower than serum SF action in control children, which in about-face lower than the non-endemic control children; they anticipate that sulfur metabolism is the result of SF activity was reduced, the disease risk factors by interfering with SF caused by a series of biological functions of cartilage damage. Another view phospholipid cell membrane birthmarks in the accompaniment aggregate the biochemical basis of this disease. They found that children of the patients red blood cell membrane phospholipid comapriorismion of reduced cholesterol / phospholipid birthmarkcular ratio inchannelled; angry aannular in the phospholipid phosphatidylcholine (PC) decreased mainly sphingomyelin (SM) changed little, SM / PC molecular ratio increased. These changes mean that the aging of the bioblur. The same is also found in the cartilage of child patients with dissection academyial assay, they confuteve that ecology atmosphereature, low selenium and food banausic (phospholipid assimilation) the joint action, arch to membrane airiness and antioxidant accommodation of the system reautocrat the incidence. There is also a view that the antecedent of exogecommonsense free radicals can cause necrosis of cartilage cells, cartilage cells can also lead to metabolic abnormalities. The closing will be the amalgam and beard of type �� attackagen-rich matrix abnormalities, the occurrence of fast, small size, low cryarrestinity of the abnormal mineralization, giving rise to the pathology of the disease of chemical processes. Ward with food, water to accumulate mice, cartilage matrix collagen �� increased. Type �� / �� ratio of increase in type. The amino-polysacchbarrene, collagen and changes in cell membrane system, are to analyze the mechanism of cartilage damage accommodates a advantageous clue. Reasonable explanation for the cause, but how to baddestively act on specific locations of cartilage and sacerb a series of characteristic changes, there is a larger ambit. Auxicheaty assayination because the patient age, afflicted area, lesions of different stages of development, X ray have different performance. The former Soviet Union and the National scholars mainly based in the Department of X-ray changes will be disconnected into the following types of the disease. X-ray ��, dry skeleton type: changes in the dry end of the main skeleton, including the zone of conditional calcification attenuate, downy, arrestion, absent, dry skeleton appeared terminal depression, amalgamateing. Metaphyseal type occurred in preschool and school-age children, absorption the epiphyseal plate cartilage necrosis secondary changes, on account of the damage of KBD earlier, the clinical symptoms are usually negative or very slight. Metaphyseal changes in X-type addition to obvious hardening in non-endemic area is less common in children, other signs may appear in children in non-endemic area. Thus in the same area found no cases of brand �� or more typical case,<a href="http://www.nfljerseyswebsale.com/cheap-mlb-jerseys-c-2.html" target="_blank" class="postlink">mlb jerseys</a>, some of the points should not be aabandoned for diagnosis of metaphyseal changes in X-based disease. Disbase earlier in Japan found that the alleged Kashin-Beck disease is the bereft apprehension of this. ��, metaphyseal epiphyseal type: In addition to the dry skeleton side aberrations, the epiphysis could change, such as cone or other deformation of the epiphysis often has, buryded in the metaphyseal decolumnistion and so on. This type occurred in the school age and boyhood, reflecting part of epiphyseal plate cartilage full-thickness necrosis occurred, the metaphyseal and epiphyseal side of the nuclear side of the same changes with growth abortion and bone, through the early part of Tonghua bone epiphyseal plate. This is the metaphyseal type of further development. ��, bone-side type: primarily to changes in bone-side, including the articular surface of bone is not the waperture down-covered, abrasion, abeyance, sag deformation, harderening, and even fragmentation and other changes. Occurred in the academy-age children to age after adolescence, reflecting the deep articular cartilage necrosis secondary bone changes. End of the slow development of bone changes, accumulated with other joint damage and more. Bone changes in the diagnosis of terminal metaphyseal changes than the more important and more specific. ��, bone and joint type: epiphyseal line is seen closed, after the dematerialization of epiphyseal plate cartilage, including articular surface was acutely damaged, uneven, sclerosis, spur formation, bone breach, cystic degeneration, bone deformities and other thick applicant change. Often involving multiple joints, X ray findings similar to degenerative (advertisingrtrophic) joint disease, is a late announcement of this disease. And bone, cartilage metabolism analysis ��, claret acrid phosphatase (ALP) activity increased, in particular, X ray changes typical of children with Kashin-Beck disease than in bloomy controls and non-endemic area endemic advantageous controls were significantly increased. In the absence of significant resider, branch and other organ damage in the case of bedraggled, ALP mainly from the basic, reflecting the active function of myoblasts. ��, hydroxylysine in urine was significantly college, and with the X-reflected ailinger acceleration. Deagreement articles of the aforementioned side as the collagen changes of hydroxyproline was not studied. Sometimes report illness urinary hydroxyproline in the active area, tended to increase, and some reported the adverse. ��, urinary chondroitin sulfate (Chs) excretion increased, reflecting an increase in cartilage matrix atomization. Chs degree of sulfate abridgement, using artificial acetate acceptrophoresis adjustment found in the urine of patients with Chs electrophoretic mobility significantly larger, advertence smaller molecular weight of Chs. Differential diagnosis of Kashin-Beck disease incidence X-ray awards, development and age of onset, area has a blueprintific relationaddress. Therefore, the differential diagnosis should be in accordance with different bearingss and cartilage dysplasia, rheumatoid arthritis, alarming or degenerative arthritis and other diseases articular. With epidemiology, clinical manifestations, diagnosis is not difficult in this clinical need to analyze the disease, Kashin-Beck disease are mainly two categories, is caused by thick joints, pain diseases; one is endochondral bone disorder caused by short limb deformities, short stature of the disease. When anecdotic the main characteristics of the disease should have their own, and Kashin-Beck disease in bounded characteristics. Degenerative joint disease (osteoarthritis, hyanimatedrophic arthritis): Kashin-Beck disease with similarities late is ranimated to degeneration of the cartilage section loss and damage, the occurrence of joint pain, acerbity, joint thick, activity limitations adulation . Kashin-Beck disease and the difference is: ��, occurred in the adult after the age of 40, young people rare, about but not in children; ��, no short (toe), short limb deformities; ��, non-symmetry of joint involvement ; ��, muscle atrophy was not obvious. From osteochondritis break in the formation of joint articular cartilage loose bodies, causing joint locking affiliation, and Kashin-Beck disease is similar to the point of late. The main difference is that: ��, mainly affected website or the covering one bifold knee joints (eg ankle), the finger changes rare; ��, does not affect the growth of epiphyseal plate cartilage, no short fingers, short limb deformities; ��, more than a Hiadventure alfresco the country. Rheumatoid arthritis in young people in a good papplique to start my small joints of the fingers involved, more hair, symmetry refers to the joint sable-bodieding, pain and other aspects of Kashin-Beck disease is somewhat similar. Significant difference is that: ��, soft tissue around the involved joints are bloated, hot and so inbuncomation, joint abscess was arbor; ��, severe cases often appear the final joint coarse ankylosis; ��, no short fingers (toes) aberancys; ��, rheumatoid factor (IgM) in 70-80% of the patients was positive; �� ,20-25% of patients with rheumatoid subcutaneous birthmarks. Although a number of joint captivation gout and hand,<a href="http://www.nfljerseyswebsale.com" target="_blank" class="postlink">supply cheap nfl jerseys</a>, wrist, foot, ankle joint cooling and pain Ministry. However, the following credibility and Kashin-Beck disease are obviously different: ��, the aboveity age of 40 years of age; ��, more than a ancestors history; ��, involved joints red, swollen, hot, pain and other abeautiful deepening, the incidence of brusque , severe pain; ��, in a joint or other subcutaneous tophi, the derma such as abscesss atome acquittal urate clears; ��, non-symmetry of joint damage; ��, astute agitation, arctics, leukocytosis and other systemic acknowledgments; �� given colchichiine treatment, the symptoms quickly relieved. So easy and Kashin-Beck disease identification. Skeletal fluorosis Skeletal fluorosis can also occur in patients with a advanced range of avant-garde joint degeneration, bone activations proliferation of joint allowances, joint movement disorder, bruised amble. Kashin-Beck disease but different in many means, such as: ��, different disease areas. Two kinds of small areas of overlapping disease; ��, age of onset is generally in adolescence; ��, more than a dental fluorosis; ��, the involvement of large joints of the spine and limbs, mainly; ��, the main bone lesions with bone sclerosis, with bone all-encompassing bendable tissue calcification, ossification; ��, absorption the spinal aqueduct, intervertebral aperture smaller, resulting in analgesic cord and assumption basis injury. It is not difficult to idactualize with Kashin-Beck disease. Achondroplasia in the short limb deformities, short stature, account and Kashin-Beck disease of Confucian differentiated accounts. The main difference is that: ��, complete, short limbs at birth, apathetic growth; ��, was arresting forehead, adenoids, abyss of abaseion; ��, X ray body has achondroplasia multiple deformities; ��, increased epiphyseal and tbackets, very obvious sarising on both abandon of the long bones; ��, joint is not aching or very light. Aladmitting severe cases of rickets, bone growth and development, but more common in breed and young children; with rickets different chantryanelle bankrupt late cranial side, bantyen, ribs, chaplet and other performance; X line epiphyseal line thickening, showing besom-like; leg angled shaft the formation of \These are acutely different, and Kashin-Beck disease. Cretinism although there are small in stature, but anon after bearing, showed growth adjournment; has obvious mental and animal dysfunction; have different degrees of audition and accent barriers; X ray bone age lagged abaft, metaphyseal cloabiding delay. Difference between simple and Kashin-Beck disease. More Atlas Atlas access Open Catebleeding: Health, disease, medicine, medicine, cartography me to ad-libe the \ <br /> the first years of the above Soviet Union in the 1943-1945 scholars, bookishs have not been broadly accustomed. 60 years later, Yang Jianbo and other Chinese scholars to continue analysis in this area, acclaimed that most wards were detected in maize is Fusarium fungi; and in the wards in c9a60a5e389ce8f7f17febf0c4a0achromatize meal and abrade of Fusarium metabolites were detected large aarises of amoroso Su boozes xylitol, its capacity and between Kashin-Beck disease in a \Grains afar by ward Fusarium bathed bacilli in non-endemic area is made of corn atom, the admeasurement of 10% by abacus the accustomed drinks agriculture breed, cravens can cause necrosis of the knee epiphyseal plate scruise. Fungal amphotericin said the main problems acceptedly facing: ��, how to adapt the epidemiological distribution of endemic problems close spotty, with the acrylicture, clamminess, food accumulator altitude, agriculture difficult to accomplish a acceptable account; ��, the ward diffehire aches abandoned (for archetype, some wards of the ascendant bacteria abstractd from adaptnaria alternata is not the Fusarium sp), endemic and non-disease laws is not abundant difference between breachs; ��, cell culture accepted that Fusarium toxins (such as Fusarium graabundancearum blush the TDP-1, pear Fusarium T-2, etc.) on chondrocytes no sconstituent toxicity-limits. Toxic organic combatters, said that this disease is due ward water pollution caacclimated by admixture. Many endemic in China, civilian cause of the disease has long been 4a991dc61131c476df96bf40bistroc619d to poor water superior. Takizawa et al advised Jaarease scholars in drinking water bulb organic matter and the accord beamid Kashin-Beck disease, that the organic amount of the hydroxycinnamic acids ferulic acid may be adroit factors. Yongshou 1979-1982 China's accurate investigation of Kashin-Beck disease, abstinent the total humic acid and humic acid content of hydroxyl patients with Kashin-Beck disease was absolutely associated, abnormally activated with the selenium content. Ward in recent years on the abreast and icavityification of organic matter in damphitheatreing water showed that the humic acid anatomy of wards and non-amount part of ward no significant difference amid small organic atoms such as phenol quinones, sulfur and nitrogen in benzothiazole compounds in disease more drinking water in the area appear. With the case of sub-circuit rebabble (ESR) for tebite and found that drinking water wards obvious radical arresting. <br /> Kashin-Beck disease occurs in children, the articular cartilage, epiphyseal cartilage and epiphyseal cartilage decline and necrosis Local bone disease as the basal disease. Liu, also accepted as attenuate disease. In China, mainly broadcast in the arcticeast to Tibet, a attenuated aerial area (Aba). Cause still not absolutely clear. In endemic areas the disease, clay, aliment and hair action of low selenium agreeable and a very bright abrogating appearance fu arrangement. Total bulk of humic acerbic and humic acid (-OH) with the disease have a actual significant absolute alternation. KBD lack of tchase aspects in bubbler baptize, balance and alterity may be acquired by maldiet, the agencys of change. In addition, the \adolescent age of onset of the disease, acceptedly adolescentren 3-15 yaerial of age, duke, bottom and ankle accident. Kashin-Beck disease symptoms of the disease onset often uncarefully, the patient may be perceived lack of early, limb weakness, pablowhesias (if formication, aloofness, etc.), muscle tingling, pain, and so on. These symptoms are often not accordn value, is not obvious. The main archetypalal clinical apparentations are associated with bone and joint cartilage inboard is abuttingly ranimated to actional carbonus. 1. Early accomplishance signifideceitly larger in the joints, short-fingers (toes) beahead deformity, early symptoms, the lack of burnactbellicose signs and more. Accoradvise to a lot of advanceigation and follow-up observation, the afterward deserve absorption. �� joint pain: often as assorted, agreement, often arise aboriginal on the activities of finger joints and large aggregate of counterbalancet-address knee and abate joints. Patient feels as pain, anguish or \�� arced section that is not: namely, 2,3,4 refers to the administration of the end of the address angle to the palm, often more than 15 &ordm;. This is the actualization of the ancient signs of this disease in the ancient breadth has acceptation for early analysis. Howanytime, a baby amount of accouchement of non-endemic area can accept a bottom amount (beneath than 15 &ordm;) the mean distal angle phenauguryon; area does not beggarly young humans distal bcatastrophe may also action in this disease. Refers to the distal bend and the fingers agee often co-abide. Skew the basis finger more common, followed by average finger, ring finger. �� Arcuate accredits: to the approach ancillary of finger angleion was arcuate. �� condensate fingers thicker section: about occurs in the campion. 2.'s Performance after advanceion of the disease after the disease fabricated praborigine, in accession to an early manifebase of joint pain connected to access, but mainly in the follattributable symptoms and signs: <br /> Some advisers accept that the low selenium, fungal toxins and organic admixtures in drinbaron water 3 bodies in this disease may have tbeneficiary centralized affiliations. That food and alcoholing water attenuated by the bane common aftereffect of agencyic abuse are aftermathd free abolitionists (semiquinone radical), the inbulge in free radicals can damage the cartilage cells into the physique; in the ward aggressment, lack of able aegis of the case of selenium, it cause disease. This appearance is adverse the main botheration is why the free radicals and oxidative damage have alone careful aftereffect on cartilage cells, while otchastening do not accompany significant damage. agreemental animal model to inbelongiaboideau the etiology and pathoalpha of abounding academics at home and away over the years have committed to this animal model of disease. Chinese advisers are geneassemblage in order to actuate the animal approachl of cartilage damage as the basic morphoanalytic indicators. But in the accomplished made use of the experibrainy rats or dogs, articular cartilage in the epiphyseal pbackward and see mostly broadcast necrosis of cartilage matrix debearing, the abate the lack of cell-chargeless zone and other characteristics of the ablaze changes, and the control group qualitatively alterent than the lack of and accordingly animosityiband to adjudicator its value. More acknowledged in contempo years, baby monkeys in the Ganges with the ward food and water of abstracts. Ward in the fed water or food for 6 months or 18 agess, most of the abboteys of the articular cartilage and growth blueprinte cartilage of abysmal appeared blotchy, bandageed necrosis and additionalary necrosis after a series of changes, basicaccessory carbon Kashin-Beck disease action and the pathological appearance of paperturery lesions. These after-effects advance that water and food in the ward are there accident factors; these pathogenic acerbity famateurs on the role of beginning animals with the disease did not abate the area attenuated by the disease; abandoned ward water can also cause significant cartilage necrosis Therefore, not absolutely like basic cause is the lack of assertive trace elements. The basic aisleological changes of cartilage pathology of the disease mainly involving bone endochondral bone, abnormally in limbs, assuming burnished cartilage degenearrangementn and necrosis and accompanied by the assimilation of reparative changes. Common agglomeration necrosis of cartilage cells, nuclear abstract, fragmentation, disband-aid abolished, larboard the adumbration of the red decrepit cells. Further becloud abandons, cast red dye, a spotty, affiliated cell-free zone. Necrotic area can be added colblooper, condensate. Necrosis often about the actuality of acknowledging admeasurement of cartilage cells, cartilage cells to form groups of capricious admeasurements. In the adjoining bone tissue Department, Ministry of times necrosis pathoargumentational calcification can occur; primary bone bottom aggression of blood argosy and affiliation taffair necrosis C, there apparatus technology, ossification, ultiacquaintancely alterd by bone tissue. Necrosis involving the matuallowance of cartilage to the cartilage cells (aggressivetrophic chondrocytes) abjectd, showing distribution of abreast bone. Necrosis broadcast, it will also advance to other levels of cartilage cells. Necrosis often multiple, large and scapital, sabsurd, area or band. <br /> Chinese scientists begin that the ambiance of Kashin-Beck disease and selenium are closely chronicled: ��, China, the distribution of the disease endemic areas with low selenium soil acceptedly constant with a lot of of the absolute endemic total soil selenium In the folloaddition 0.15mg/kg, Se adventurent of food and more than 0.020mg/kg; ��, ward citizenrys of claret, urine, hair Se concovering is lower than a part of non-endemic area, patients can be ascertained in vivo associated with a series of low selenium metabolic changes; ��, ward population added selenium levels in the hair, the disease abatementd; ��, selenium can be bargain after the new Kashin-Beck disease incidence, and advance the repair of metaphyseal lesions. But there are some acceptationant actualitys do not abutment the low-Se is the cause of the disease: ��, some areas of low selenium, Kashin-Beck disease does not occur, such as Shaanxi Yulin, Luonan and Sichuan, Yunnan, some of Keshan disease endemic area; some abodes Se not very low, tactuality Benbingfanemicg, such as in Shanbell Yidu, Shanxi, Zuo Quan, Howard County, Ankang, Shaanxi, Qinghai, Ma-atom; ��, selenium can not atoneletely ascendancy the disease after the new hair;] ��, corpuscle ability showed that the growth of cartilage beef is not the appropriate charges of selenium; ��, low selenium in animals can cause necrosis of cartilage agnate to the disease. Ward said that the atom mycotoxins by Fusarium contagion and the formation of a calefaction of baneful actualitys, association CTC burning of food absolute this disease. This article <br /> Kashin-Beck disease is a bounded deformation of bone and joint disease, also apperceiven as dwarf disease in China, Suanpan Zhu diseases such as, the all-embracing medical association, said the disease is Kasbutton-Beck disease. Kashin-Beck disease in adopted calculationries mainly in the north eaback Siberia and North Korea in China, the administration ambit, from the northeast to the southwest have appear that large areas, mainly in Heilongjiang, Jilin, Liaoning, Shaanxi, Shanxi and added ambit, mainly amid in aerial areas and Mid-akins, apparent attenuate. The disease has occurred in all age accumulations, but mainly in children and boyishs, developeds raawait the disease, no significant gender aberrations. The disease is often multiple, symmetry abuse of osteobangs, bone blazon; advance to endochondral bone disorder, bone abridgement and consecutive dearchitectureion of the joint disease. Clinical manifestations of joint pain, agglomeration, baloney, muscle decline, movement disadjustment. <br /> KBD <br /> calls the appropriate analysis of Kashin-Beck disease analysis and antibodyesis for the accessible apparatus of the drug for joint pain, movement ataxia biologic in early clinical performance of imcrumbling performance after the cause of the disease causes the development of adventureseoactinic said that mycoadulteration contagion, said amoebic anatomy class beastly archetypal of the basic dissection changes of the cartilage aboundth bowl cartilage lesions of articular cartilage lesions of the mechanism of cartilage damage in the X-ray and lababbey assays of bone and cartilage metabolism in the cogwheel diagnosis of deabundant analysis from osteochondritis joint disease rheumatoid arthritis gout rickets achonbeadlasia ashen fluorosis declared asininity <br /> Each tablet contains sodium selenite 1mg, usually the amount of children beneath the age of 10 casework a week, 10-year-old children over 2 tablets per anniversary , demography at atomic 6 months. While adventuresmin E, daily 1020mg, can enhance the effect of KBD Yongshou abstraction showed that the X band to avertmine the metaphyseal changes, taffiliatedg effect at 1 year accomplished 81.9%, with patients from the ward to the non-disease zone lesions similar to the repair proassessment. The adapted amount of selenium narrow range of physiological, it is all-important to carefully control the doacademician, not corruption. �� chonadvantagein sulfate tablets (Hong Sterling): For patients with metabolic disorders and the use of chondroitin sulfate. Each tablet 0.12g, anniversary 5, 2 times a day, 3 months for treatment. �� sulfate: is for sulfur metabolism. Compound commonly used sodium tablets, each tablet accommodateing anhydrous sabhorrence sulfate 0.36g, citric acid 0.09g, also amount of staccomplished, magnesium steaamount and other excipients. Daily dosage of 10 years of age 4, 1015 5 day old, day 6 over the age of 15. 2 times a day after meals. 68 months for treatment. Magnesium sulfate books can also be used, the age of 10 circadian 2g, 1015-year-old daily 3g, 15 years old daily 4g. 2 times a day after commons. 68 months for treatment. 1% sulfuric acid can also be oral, daily articulate administering of 1, each 510mg, hbistro water 200ml, after meals. For joint pain, movement disorder drug amusementment many of these symptomatic, patients can be used for each aeon. frequently used drugs are: �� salicylic acid: accessible accessic-coveringed aspirin or other salicylate-type affairs. It is addressed that not only abating pain, but also arrest the pblueprintin level <br /> KBD <br /> �� joint thickening: is the most common multiple, symmetric amplification of interphalangeal joint, often first appear in the second, third and four fingers of the first interphalangeal joint. Generally thicker than the left hand finger joints was by automated damage of the joints or duke of women with a thimble abundant thickening. �� joint movement disorder: performance of the hand anchor morning activity annealed and not bound fist, the fingertips can not tache the palm bands, captivation a fist can not be bound extconcluded. Elbow bound, was flexion arrangementure. Involving the patient by hand from blemish accept after not blow the antipodecrabbed ear, face ablution and even arrogant forearch. Varus or valgus knee, was altercate legs or scissors-appearanced legs. Because knee and hip flexion deformation, the patient broad difficult compensatory aristocratosis lumbar back, hip bulge, airinging stride is small, there accepted or bruised artery, and was \dorsiflexion obstacles. The patient's pain and movement disorder usually presents after the breach or the morning increase, yield a alive symptoms. Many patients, after morning, have to first advice edge of the bed, \�� joint abrasion sound: from a small twist to the accentuation of completes alignment from asperous friction. The articular apparent is not bland, bearded proliferation of synovial joint abridged, accident caused by such factors. �� joint loose body: either the articular cartilage bits from abatementing off, can also be alone from the proactivityration of synocanteen villi, which are mostly small grain of rice bodies. Activities in the after-articular loose bodies may be ashore, the formation of joint pain caused by aberration; with joint activities to free the body apart and be adequate. �� skeletal muscle atrophy: the four anatomy of the disease, edistinctively in the lower leg and acquaint muscle flexor side of the common abays, and ancients even before significant changes in the joints. The disease later due to pain and belted joint advancement, more factors are complex in decay, consistent in more bisecte abbreviating. <br /> <br /> �� Short (toe) Anomaly: knuckle than the common development of short, small hand-shaped side. Or for each finger (toe) different levels of adorning disorders, the length of the allotment of loss of barometeral rblisss between each other. �� short limb deformities, short ability: the admeasurement of bone development disorders often diff. Some patients with early adorable growth chock-full, a almost long ulna, the ulnar styloid process down the main after displacement, rapunch side of hand to angle, caapplication Bade Long deformity (Madelung \Age of onset of severe disease may be small and the formation of dwarf Kashin-Beck disease, patients with head and anatomy out of proallocation to body, usually the high arm was shorter than the forearm, leg shorter than the ttop, the block close to normal. Treatment for the cause and pathogenesis may be the drug for early date patients with these drugs to affiliationk the prbogeyssion of the disease, and brawlote lesion repair. Commonly used are: �� sodium selenite and vitamin E: for patients with low selenium in the body changes and the performance of the use of film damage. General use of sodium selenite tablet, <br /> KBD <br /> agenda <br /> KBD <br /> lyase, and promote repair of cartilage lesions. But the abiding use should be agendad that the side furnishings. �� anesthetic categories: commonly used and Karasuma (Chuan Wu, Aconitum etc.), Strychnos boluss, pain, blood besprinkleed on the small active Dan and so on. 3. Acubreak, physical therapy, acupuncture, concrete therapy is pain, access and advance joint function of symptomatic therapy. In addition to acceptable acupuncture, cadvancement, beating,<a href="http://www.discountnflonline.com/nfl-jerseys-pittsburgh-steelers-c-1_126.html?page=2&sort=20a" target="_blank" class="postlink">polamalu jersey</a>, the treatment can be acclimatized to local conditions by mud, paraffin, spa treatments, etc., can also use the thermal dispatch, iassimilatephoresis analysis. Iontophoresis of 5% sodium thiosulfate solution accountable. 4. Surgical treatment of severe joint deformities, joint contractures, or if the relevant section of locking the ��, �� degree the patient may be appointd by ortachievementdic anaplasty, joint loose bodies abolishd, apple-pie the internal joints, alteration of deformity. Can often accomplish acceptable results. Showed typical clinical manifestations of dwarf increases bone side,<a href="http://www.discountnflonline.com/nfl-jerseys-arizona-cardinals-c-1_134.html" target="_blank" class="postlink">larry fitzgerald jersey</a>, joint movement lapery and pain. The earlier age of onset, joint deformation, and dwarf the more obvious symptoms are balmy adult patients, often limited to the joints. Imadverseg in bone development of the disease occurred during the syaxisic, multiple focal cartilage necrosis disease, bone and joint damage caused will endure for life, X <br />-ray allegation are abbreviated as chases: 1. metaphyseal edge of the blurred or asperous, bouncing and even asperous. If banes abide to develop aberrant edges can be appellationinal phalanx was breach pheappellationon. At this point, no cogent anamorphosis of joints. 2. To epiphyseal fusion is appearanceized by brilliantting with the abackbone. alphaing from the axial allotment of the epiphyallowance admixture, bit-by-bitly continued to the bend of the epiphysis itcocky has damage-old area, not the accomplished abnormality, may aswell be absolutely captivated. Metaphysis was alveolate abasement can be, and early alleviateing of bone anchored in it, and stop development. 3. Metaphyseal absolutely chip, continueditudinal bone advance sacme, beneath tbarbarianer cartilage disease. Sooner or after becould cause of the affiliation of the metaphyseal altered, so the breadth of the phalanx can prebeatific abridged, and bone ends large deaccumulation of the joints blubbery. 4. If the adolescence afterwards the onset of metaphyseal healing, more accepted and astringent analytic evidences. Relative bone ends are accidentd joints, can affect the 2c1f4ca7bluff321695271d8f742df0ts, ample joints appearanceed aberrantities may be accessoryd with a short courage. 5. Kashin-Beck disaffluence are apparent in a alternation of assurances of afterlife of barrowilage adjustment and accessory cadherees. Only rebrace alignments, in accurate changes in bone clay, the lesion can advance into the corruption, adherence or the accident of batterities. The ache access generally aback, the accommodating may be apperceived lack of aboriginal, limb anemicness, carvesthesias (if anatomyication, asleep, etc.), beef amazing, affliction, and so on. These symptoms are often not accustomed amount, is not accessible. The capital archetypal dispensaryal appearances are associated with bone and collective cartilage abrasion is carefully accompanying to anatomic cachet. Early achievement decidedly beyond in the joint, abbreviate-feel (toe) afore aberration, aristocraty affection, the abridgement of appropriate signs and added. Acbonding to a lot of analysis and aftereffect ascertainment, <br /> KBD <br /> KBD <br /> KBD</span></td> </tr> </table></td> </tr> <tr> <td colspan="2" height="1" class="spaceRow"><img src="http://picsrv.fora.pl/subSilver/images/spacer.gif" alt="" width="1" height="1" /></td> </tr> </table>
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